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Carbofuran ban is good for everyone - in USA only

Category: Pesticides, carbofuran | Date: Nov 09 2009 | By: paula

This really good article from Tree hugger explains the benefits of the carbofuran ban in USA

As of the end of the year, one more pesticide will be absent from food crops grown in the United States.

In May the EPA ruled that the current residue limits of the insecticide carbofuran on food crops was too high, and the agency has now decided to fully revoke carbofuran tolerances (more commonly known as residue limits). What this means is no carbofuran residue on a food will be deemed acceptable as of 2010. The move follows in the footsteps of the European Union, which banned carbofuran nearly a year ago. But the U.S. ban isn’t all that surprising–it has, after all, been three years in the making.

What Is Carbofuran?

Carbofuran is a white crystalline solid insecticide used to control nematodes, rootworm, and beetles. It is sprayed on soil and plants, just after the plants emerge from the ground. Carbofuran is used on a number of crops, including alfalfa, rice, grapes, and corn.

While there is no evidence to suggest carbofuran is carcinogenic, the World Health Organization has determined carbofuran a cholinesterase inhibitor, which means it blocks neural transmissions.

The health effects of short-term exposure to carbofuran include headache, sweating, nausea, diarrhea, chest pain, blurred vision anxiety, and muscle weakness, all of which can be reversed, according to the EPA. But the long-term effects are far more serious: permanent damage to the nervous system and the reproductive system.

For the average person who does not work with carbofuran, exposure routes include both residues on foods and drinking water contamination from farm runoff.

Cabrofuran is also a problem for wildlife. Earlier this year, reports emerged that carbofuran is responsible for poisoning of African lions.

The Benefits of Going Carbofuran-Free

The move will minimize risks to agricultural workers and the environment, but it will also improve food safety, says Steve Ownes, assistant administrator for EPA’s Office of Prevention, Pesticides and Toxic Substances:

The evidence is clear that carbofuran does not meet today’s rigorous food-safety standards. [The] EPA has carefully evaluated the scientific issues and has provided more than 500 days of public comment on this decision. It is now important to move forward with the needed public health protections, especially for children.

The move also helps keep carbofuran out of fresh water sources, which has been on the EPA radar.

Carbofuran Cancellation Timeline

The move to revoke carbofuran residue limits was a long and careful process that weighed the risks against the benefits of using the insecticide.

In 2006, the EPA identified considerable dietary, occupational, and ecological risks related to the use of carbofuran. The agency decided the risks outweighed the benefits of using the pesticide, and set out to cancel the use of the pesticide.

In January 2008, the EPA submitted a draft Notice of Intent to Cancel use of carbofuran to the Federal Insecticide, Fungicide and Rodenticide Act (FIFRA) Scientific Advisory Panel and the United States Department of Agriculture (USDA). Following review from the FIFRA panel and the USDA, the EPA decided to move forward with canceling the use of carbofuran.

In March 2009, FMC Corporation, which produces carbofuran, voluntarily canceled uses, with the exception of use on field corn, potatoes, pumpkin, sunflowers, pine seedlings, and spinach grown for seed. Artichokes were supposed to be given a two-year phase-out period.

On October 30, 2009, the EPA announced all crops would be subject to the December 31, 2009 deadline for revoking carbofuran tolerances, doing away with previous phase-out plans.

According to an EPA press release, the agency is currently encouraging growers to prepare to switch to “safer pesticides or other environmentally preferable pest control strategies,” adding that carbofuran should not be applied to food crops after the end of the year, in order to comply with the new standards.

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3 year old Child dies after eating Furadan in Kenya

Category: Pesticides, carbofuran | Date: Nov 06 2009 | By: paula

Dear friends,

We can confirm the tragic reports of a human death due to carbofuran poisoning. Just today we spoke on phone with the heartbroken father of a child who died of Furadan poisoning. The report of this death first appeared on Kenya’s The Standard newspaper on Friday, 30 October 2009 saying that on Monday, 26 October 2009, the child had mistakenly ingested Furadan and died.

The child’s father informed us that the child died on arrival at the Cherangani Nursing Home in Trans Nzoia East District in western Kenya. The father had bought the pesticide four months ago for use in killing insects in the soil when preparing his vegetable nursery. He says that he was not aware how dangerous the product is and was not informed by the retailer about the first aid approach in case of pesticide ingestion. He gave his child milk and crushed eggs - a method of dealing with poisoning widely used in Africa - instead of water as the label says.

This tragedy could have been avoided - the father, an educated man  (he is a teacher at a local primary school) did not get the impression that this pesticide was deadly. The packaging in kenya does not carry teh universal symbol of death - the skull and crossbones.

Please join us in sending our sincere condolences to the parents of 3 year old Kimutai, and pray that he rests in peace.

We hope that Kimutai did not die in vain and that the Kenyan government takes appropriate action by baning carbofuran in Kenya immediately.

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10 more lions poisoned in Masai Mara

Category: Masai Mara, Pesticides, carbofuran, lions | Date: Nov 01 2009 | By: paula

WildlifeDirect has been raising the alarm about cattle in Kenya’s parks for some time know - cattle grazing in the park will lead to conflict with lions and this has an inevitable outcome. CAttle will die and then lions will be killed  in retaliation.  We warned of diseases when cattle started dying in the parks, and Dino wrote about it in his blog dudu diaries here.  The authorities ignored our comments and concerns about the cattle invasion when we warned that an Anthrax outbreak would affect cattle, wildlife and people.  I even went on radio about it and finally it seems, people woke up and began to listen.

In a recent article in the Daily Nation, the Kenya Wildlife Service (KWS) admitted that

A total of 10 lions have been killed by herders who have lost their cattle to the large cats. In one case, farmers poisoned a carcass and it left for the lions. It killed not just a lion, but also 300 vultures that ate the carcasses of the cow and the lion”.

No doubt these ten lions were from one pride and were poisoned. The loss of 300 vultures suggests that the poisoning was widespread - this could not have been just one incident.We will try to get details on what actually happened and determine if carbofuran was to blame.

The head of the Species Program, Mr Omondi,  warned herders that they should expect to lose some of their animals if they choose to break the law and let them graze in protected areas.

The problem of livestock in game reserves which WildlifeDirect raised on Kiss FM Radio as well as through our blogs is so serious that it has consumed the greatest budget line for KWS during recent months.

The KWS says that the greatest challenge it faces is that in Reserves like the Maasai Mara, the management authority, the Narok County Council, turns a blind eye to the herders. Sadly the same is true of KWS who have for years allowed grazers into parks during annual dry seasons.To date we do not know of a single herder that has been prosecuted for illegal grazing, or of poisoning lions, vultures, hyenas or other animals.

This story reveals just how difficult it is for KWS to control the situation and protect Kenya’s lions.  Lions live mainly in areas that are not under KWS control. Many protected areas are poorly managed. There is nothing stopping herders from entering parks and reserves - even if one is caught, there are no penalties. Moreover, there are no incentives for communities to protec t lions and other wildlife outside of the protected areas. When  livestock are killed, the KWS is supposed to compensate owners, but this is a lengthy and controversial if not poorly managed process. Add tho this the easy access to pesticides like deadly carbofuran and any pastoralist can solve the problem of predation in an instant. Just a few granules of the purple killer will deal with an entire pride plus any other stragglers or plike hyenas.

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USA: zero tolerance for carbofuran - its not safe

Category: carbofuran | Date: Oct 30 2009 | By: paula

Dear all We are pleased to share this press release today from the EPA  

WASHINGTON – The U.S. Environmental Protection Agency is moving forward to implement the agency’s May 2009 final rule revoking tolerances, or residue limits, for the pesticide carbofuran. EPA continues to find that dietary exposures to carbofuran from all sources combined are not safe.

“The evidence is clear that carbofuran does not meet today’s rigorous food-safety standards,” said Steve Owens, assistant administrator for EPA’s Office of Prevention, Pesticides and Toxic Substances. “EPA has carefully evaluated the scientific issues and has provided more than 500 days of public comment on this decision. It is now important to move forward with the needed public health protections, especially for children.”

Short-term health effects include headache, sweating, nausea, diarrhea, chest pains, blurred vision, anxiety and general muscular weakness.

EPA encourages growers to switch from carbofuran to safer pesticides or other environmentally preferable pest control strategies. Since the tolerances are being revoked, EPA reminds growers that carbofuran should not be applied to any food crops after December 31, 2009. Use of carbofuran after this date could result in adulterated food products, which would be subject to enforcement by the U.S. Food and Drug Administration. The United States has a safe and abundant food supply, and everyone should continue to eat a variety of foods, as recommended by the federal government and nutrition experts.

During the objection period, the chemical company FMC Corporation, which manufactures carbofuran, and three grower associations submitted objections to EPA’s tolerance revocations and requested an administrative hearing. EPA has concluded that the regulatory standard for holding an evidentiary hearing has not been met.  EPA’s explanation about why a hearing is not warranted, and the reasons for denying the objections are available on the web and will be published soon in a Federal Register notice.  As part of the administrative process, FMC may appeal the revocation of the carbofuran tolerances to a federal circuit court of appeals.

EPA’s May 2009 action to revoke carbofuran tolerances was the culmination of a regulatory process that began in 2006 when the agency published its risk assessments for carbofuran and determined, in August 2006, that no uses were eligible for reregistration. While FMC has voluntarily canceled 22 carbofuran uses, the elimination of these uses was not sufficient to allow the agency to make a finding that combined dietary exposures to carbofuran from food and water are safe. The process to cancel the remaining carbofuran registrations is under way and will address unacceptable risks to farmworkers during pesticide application and to birds in and around treated fields.

More information: http://www.epa.gov/pesticides/reregistration/carbofuran/carbofuran_noic.htm

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5 lions poisoned in Serengeti

Category: Masai Mara, Pesticides, carbofuran, lions | Date: Sep 18 2009 | By: paula

Dear Friends, this report is just in from Arusha from a colleague. We will get photos shortly.

“I met a guy who confessed that Furadan has been recently (2weeks ago)
used to kill 5 lions around Serengeti. TANAPA are investigating and
they have taken samples of the dead lions to establish the actual
poison. The story was; the lions killed a giraffe near a maasai boma.
The Maasai, fearing the lions would attack their livestock after
finishing the giraffe, they laced the remaining giraffe carcass with
furadan. That evening the lions came back and 5 of them were found
dead near there the following morning!! There is a lot of Furadan in
Arusha. I bought a 500 gms from the Tanzania farmers association shop
at an equivalent of $15. Kisamo (TANAPA) promised he would share the
lab findings of the samples once they are out. He will also send us
the photos when he gets them from the guys who went to the ground when
the incident occurred! I am sure we won’t win the battle if Tanzania
still has the furadan distributed by JUANCO from Nairobi.”

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Huge Bird deaths in Thika, Kenya

Category: Uncategorized | Date: Aug 30 2009 | By: paula

We have just recieved reports that there has been a huge die off of birds at the Thika sewage works just north of Nairobi. This sewage works has been a favourite place for birders as it attracts a huge diversity and massive congregations of birds local and migrants

bird deaths thinka kenya

A team from Ornithology dept NMK in the company Oliver Nasirwa went to the Thika
sewage ponds to assess the reported case of dying birds at the site on the 26th
August 2009.

poisoned duck kenya

Ronald Mulwa notes:

“From my assessment and talking to the officers on the ground, the die off cases could
be going down. We found one Sacred Ibis really sick and unable to fly, also found one
Red-billed Teal just dying - apart from that the rest were 1 week old (or so) carcasses -
we assume that some carcasses also get swept away into the sewage outlet.

Though we are working on a more detailed update, the following are the
birds we found dead:
Sacred Ibis - 2 + 1 unable to fly
White-faced Whistling Duck -1
Red-billed Teal - 15
Red-knobed Coot - 5
Hadada Ibis - 2
Black-winged Stilt - sickly and unable to fly 1

black winged stilt poisoned

We thought this may not be termed ‘Mass Die Offs’ as such, since there were still 100s of birds feeding and actively flying around. But the root cause for the deaths need to be established urgently.

We took samples some carcasses that were in reasonable shape and have been taken to
Kabete Vet Labs this morning. The Cape Teal we found dying had a strange swellings ballooning out of both eyes like bubble! photos available!

The officer in charge was quit concerned, supportive and was keen to be involved in this
assessment and to see the results of the Lab analysis.

We welcome suggestions and further discussion.
Best regards
Mulwa Ronald
Research Scientist Head - Ornithology Section, Zoology Department
National Museums of Kenya
P. O Box 40658 00100
Nairobi Kenya
Tel: 254-20-3742131/3742161 extn 243
Fax: +254-20-3741424 Cell Phone: +254 722499

According to Brian Finch and a report from Oliver Nasirwa of Nature Kenya, the three days between the initial discovery on 23rd August 2009 and Olivers visit three days later, there was incredible variation in what both parties recorded.

Some of the dead birds disappeared including fifteen dead Spur-winged Plovers, Yellow-billed Ducks, Hottentot Teal, several Ruff and more than five Coot, is a mystery. This could be due to scavenging animals are moving in from the surrounding farmlands, maybe even local dogs.

Brian notes “the difference in live presence which is amazing, our figures
in brackets:

Little Grebe 450    (250)
Sacred ibis 170     (6)
Cattle Egret 5     (nil)
Yellow Billed Stork 13  (1)
Yellow-billed Duck 30   (15)
White-faced Whistling Duck 30   (20)
Red-knobbed Coot 50 (75)
Egyptian Goose 60  (40)
Grey Crowned Crane 12  (4)
Black-winged Stilt 100  (60)
Spur-winged Plover 50  (4 live fifteen dead!!!!)
Common Sandpiper 20 (20)
Curlew Sandpiper 30 (5)
Wood Sandpiper 10  (70)
Marsh Sandpiper 6 (1)
Little Stints 70 (90)
Chlidonias terns 30  (1WWBT)

We also recorded 2 White-backed Duck,  8 Hottentot, 2 Glossy Ibis, 10 Hadada, 2 Long-toed Plover, 6 Blacksmith Plover, 15 Three-banded Plover, 50 Ruff, 2 Green Sandpiper.

I think it erroneous to assume that birds that appear perfectly healthy are not infected and succumb later. Also I think that the difference by the two counts testifies that there is a considerable movement through the ponds, but even the birds that move on south or
wherever could have taken in a fatal dose.

If this were a terrorist situation we would be on a RED not ORANGE alert!”

The hotline to report bird die-offs to the Department of Veterinary Science is 0722-726-682.

To join the Nature Kenya bird group email kenyabirdsnet@yahoogroups.com

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Masked Long-term effects of Pesticide Toxicity

Category: Pesticides | Date: Aug 13 2009 | By: Martin Odino

University of Pittsburgh researchers have reported in the September edition of Environmental Toxicology and Chemistry that the four-day testing period the U.S. Environmental Protection Agency (EPA) commonly uses to determine safe levels of pesticide exposure for humans and animals could fail to account for the toxins’ long-term effects.

Environmental Toxicology and Chemistry article reports “lag effect,” revealing that harmful effects can remain hidden until after EPA’s four-day direct exposure test.

The likes of neurotoxin endosulfan can exhibit a “lag effect” with the fallout from exposure not surfacing until after direct contact has ended. Lead author Devin Jones, a recent Pitt biological sciences graduate, conducted the experiment under Rick Relyea, an associate professor of biological sciences in Pitt’s School of Arts and Sciences, with collaboration from Pitt post-doctoral researcher John Hammond. In the study, the team exposed nine species of frog and toad tadpoles to endosulfan levels “expected and found in nature” for the EPA’s required four-day period, then moved the tadpoles to clean water for an additional four days, Jones reported. Although endosulfan was ultimately toxic to all species, three
species of tadpole showed no significant sensitivity to the chemical until after they were transferred to fresh water. Within four days of being moved, up to 97 percent of leopard frog tadpoles perished along with up to 50 percent of spring peeper and American toad tadpoles.Read more on the “lag effect” here.

Relyea explains that endosulfan is 1,000-times more lethal to amphibians than other pesticides. Yet, he said, if the
powerful insecticide cannot kill one the world’s most susceptible species in four days, then the four-day test period may not adequately gauge the long-term effects on larger, less-sensitive species.

“When a pesticide’s toxic effect takes more than four days to appear, it raises serious concerns about making regulatory decisions based on standard four-day tests for any organism,” Relyea said. “For most pesticides, we assume that animals will die during the period of exposure, but we do not expect substantial death after the exposure has ended. Even if EPA
regulations required testing on amphibians, our research demonstrates that the standard four-day toxicity test would have dramatically underestimated the lethal impact of endosulfan on even this notably sensitive species.”

Andrew Blaustein, a professor in Oregon State Universitysaid the results raise concerns about standards for other chemicals and the delayed dangers that might be overlooked. “The results are somewhat alarming because standards for assessing the impacts of contaminants are usually based on short-term studies that may be insufficient in revealing the true impact,” Blaustein said. “The implications of this study go beyond a single pesticide and its effect on amphibians. Many other animals and humans may indeed be affected similarly.”

Tadpoles in the Pitt project spent four days in 0.5 liters of water containing endosulfan concentrations of 2, 6, 7, 35, 60, and 296 parts-per-billion (ppb), levels consistent with those found in nature. The team cites estimates from Australia-where endosulfan is widely used-that the pesticide can reach 700 ppb when sprayed as close as 10 meters from the
ponds amphibians typically call home and 4 ppb when sprayed within 200 meters. The EPA estimates that surface drinking water can have chronic endosulfan levels of 0.5 to 1.5 ppb and acute concentrations of 4.5 to 23.9 ppb.

Leopard frogs, spring peepers, and American toads fared well during the experiment’s first four days, but once they were in clean water, the death rate spiked for animals previously exposed to 35 and 60 ppb. Although the other six species did not experience the lag effect, the initial doses of endosulfan were still devastating at very low concentrations. Grey and
Pacific tree frogs, Western toads, and Cascades frogs began dying in large numbers from doses as low as 7 ppb, while the same amount killed all green frog and bullfrog tadpoles.

The endosulfan findings build on a 10-year effort by Relyea to understand the potential links between the global decline in amphibians, routine pesticide use, and the possible threat to humans in the future.

A second paper by Relyea and Jones also in the current Environmental Toxicology and Chemistry expands on one of Relyea’s most notable investigations, a series of findings published in Ecological Applications in
2005 indicating that the popular weed-killer Roundup® is “extremely lethal” to amphibians in concentrations found in the environment. The latest work determined the toxicity of Roundup Original Max for a wider group of larval amphibians, including nine frog and toad species and four salamander species. For more, please read the Roundup paper.

In November 2008, Relyea reported in Oecologia that the world’s 10 most popular pesticides-which have been detected in nature-combine to create “cocktails of contaminants” that can destroy amphibian populations, even if the concentration of each individual chemical is within levels considered safe to humans and animals. The mixture killed 99 percent of leopard frog tadpoles and endosulfan alone killed 84 percent.

A month earlier, Relyea published a paper in Ecological Applications reporting that gradual amounts of malathion-the most popular insecticide in the United States-too small to directly kill developing leopard frog tadpoles instead sparked a biological chain reaction that deprived them of their primary food source. As a result, nearly half the tadpoles in the experiment did not reach maturity and would have died in nature.

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Furadan used to kill moles in Baringo

Category: Masai Mara, Pesticides, carbofuran, lions | Date: Jun 03 2009 | By: paula

A colleague dropped in to our office to tell me that the buy back program was working well in Baringo where farmers were up in arms because they can no longer  purchase their favourite ‘mole killer’.

Listen to this interview  and send us your thoughts.

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Voice of America on Furadan

Category: Masai Mara, Pesticides, carbofuran | Date: Jun 02 2009 | By: paula

This article has just been published on Voice Of America and aired on radio here

As Wildlife Dies, Kenya Considers Pesticide Ban
02 June 2009  
[insert caption here]
Lion poisoned by Furadan - WildlifeDirect

In Kenya, parliament is being asked to ban a pesticide that’s been blamed for the deaths of hundreds of animals, including many lions. Kenyan MP John Matutho is introducing legislation to prohibit the use of Furadan – a cheap but lethal chemical originally manufactured by the US based FMC Corporation.

The conservation group Wildlife Direct supports the ban, which would replace a buy-back program for Furadan. It says local herdsmen are using it to poison lions and other carnivores threatening their livestock.

In Nairobi, WildlifeDirect executive director Dr. Paula Kahumbu says, “This is a pesticide that has recently been banned in the United States. It’s also banned in Europe because it’s been found to be unsafe to be used even if we follow the label instructions.… It’s one of the most dangerous pesticides actually available at the moment.”

It’s readily obtainable over the counter in Africa. “It’s very, very cheap. In fact, it’s probably the cheapest pesticide available,” she says.

Deadly to wildlife

In the early 1990s, it was discovered that water birds were dying large numbers after Furadanwas used in some irrigation systems.

“So that’s when people realized it was just devastating wildlife. And later on, the local communities realized it was powerful against almost any animal. In fact, birds are very sensitive, but so are cats,” she says.

Lions fall prey to Furadan

“We know over 60 lions that have been killed in the last two years and that’s probably the tip of the iceberg. And Kenya today has fewer than 2,100 lions remaining. We used to have over 30,000,” says Kahumbu.

Is the buyback plan working? The head of WildlifeDirect says, “The Furadan withdrawal and buyback is working in the sense that FMC is effectively withdrawing it from the shelves. The problem is the patents that FMC had have expired and Furadan, or carbofuran, is being produced now by Chinese, Indian and Pakistani companies.”

The conservation group fears that means unless a ban is imposed, the pesticide will easily find its way back to Kenya.

The chemical attacks the nervous system and only small amounts can kill an animal. It can also be fatal to humans if ingested.

“It takes only a quarter of a teaspoon to kill people, “says Kahumbu. She says lower concentrations can cause neurological problems, such as paralysis and breathing problems.

“This has been documented in other countries. It hasn’t been documented in Kenya. And I suspect it’s purely because there’s absolutely no monitoring system in place,” she says.

Enforcing a ban

“It’ll be easy to enforce in that if anybody is found using it there would automatically be very stringent responses. People would be arrested. They probably would be fined or maybe even go to jail,” she says.

Once a ban is imposed, she says, an education campaign can begin warning of the health dangers of Furadan and the risks of punishment for using it.

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A poisons’ (especially Furadan’s) weekend!

Category: Pesticides, carbofuran | Date: Oct 05 2008 | By: Martin Odino

I hope you all had a wonderful weekend!

Apologies if this post’s heading is troubling; I could not find any better title. I also wish to humbly inform my dear readers that I must leave certain organization’s names out so that this does not turn personal. I was not comfortable when somebody that matters in one pesticide organization told me, ‘welcome! I have heard about you and I am glad I have seen you’.

I went through a turbulent end of the week! I literally spent the Friday and Saturday struggling in my limited ways together with one remarkable conservationist heading the Kenyan office of a renowned international conservation organization, trying to get the details of recent Furadan poisoning of fish in Tanzania. I am still optimistic that I will get some details and hopefully, photos sent my way across the border (from Tanzania) of the poisoning ordeal. I should then surely avail the story on the stopwildlifepoisoning blog. At the moment, I only know that 6 gallons of liquid Furadan were poured in Kilombero River to kill fish with the fishermen warning the villagers not to use the river water for domestic purposes. This happened sometime last week.

I spent the weekend combing papers and articles on carbofuran especially Furadan 5G, the grossly alleged threat to Kenya’s wildlife. I must get certain facts right to be able to tackle the various troubling facets that challenge this stop wildlife poisoning campaign, in particular the campaign against Furadan pesticide as the poison. These have been manifested in my interactions especially with the people dealing with pesticides before it gets to the users.

It is now apparent that the discussion about Furadan is not a discussion but a sad war. It has become tough for me to get any relevant information from the pesticides fellows. It starts with word games where we have been repeatedly warned that we should talk of Furadan poisoning and not Carbofuran poisoning despite the active ingredient being carbofuran. I was recently advised for my knowledge that carbofuran is not sent nor sold in Kenya, but as far as I know, it is sold and sent to Kenya in the Furadan preparation. Nonetheless, I am going to stick to Furadan poisoning to save myself from the inconvenience of being interrupted and getting confused from the flow of my conversation that I should not mention carbofuran poisoning but Furadan poisoning. That is not the end of the war, I am disappointed when I am directed to a website where I cannot find information especially concerning a follow up on a Furadan alleged case of poisoning (Mara lion poisoning). Either the information is not there absolutely, or it is hidden behind the locks of a registration fee that is required for anyone to have full success of the information on the website. I wonder why positive counter allegation evidence to an issue that sparked terror and implicated a great need to mend holes in the pesticide regulation /manufacture fraternity would be kept hidden from the public. Many questions therefore arise as to the credibility of the findings of the follow up which was summarised as ‘there was no connection between the dead animals and carbofuran’ in the Mara.

I have also been trying to find out the carbofuran products that may have been or are still of concern in other places in the world in trying to establish if I can link it up to the Kenyan scenario. Based on a report in late 1990’s-crop-profile-of-rice-in-california.pdf- I stumbled on a profile description of Furadan 5G, the exact carbofuran product that may cost Kenya its wildlife and probably aggravate the neurotic disorders of its citizens. Various aspects of Furadan 5G are highlighted including its safety. According to the report, carbofuran was on the Food Quality Protection Act list 1 of insecticides scheduled to have their tolerances reassessed by August 1999. As a carbamate, the report revealed that the reassessment of carbofuran may result in the elimination of some uses. The product seems to have been praised for its minimal effects on non-target arthropods and fish. This is not what we are experiencing in Kenya, or are we dealing with a compound pseudo-labelled Furadan 5G when the reality is that it is a higher concentrate carbofuran product? Our Furadan 5G product even has one of the hazard labels cautioning on harm on fish. It does not make sense when it is generally stated in a communication to me that Furadan 5G is generally less toxic than the active ingredient carbofuran by 20-40 times. Fish were poisoned last week in Tanzania and birds, also fish are still being poisoned in Kenya using Furadan 5G. These have ended up and will continue ending up in East African peoples’ digestive systems, the actual effects on their health of which need the medical personnel to unveil.

I also gathered from an International POPs Elimination Project report of 2005– that spelt doom due to the hazardous state attributed to many chemical stockpiles including poor storage of which Desert Locust Control (DLCO) East Africa was sited. DLCO particularly struck me because it was reported that they had switched from mostly organchlorines which were banned due to persistence, environmental effects and bioconcentration in fatty tissues, but switched to amongst others carbamates carbaryl and propoxur. These are less hazardous to the environment but more acutely hazardous to human and animal health. When these poisons are injected into the air, they will not only just bring down the insects (starting with locusts) but also birds (directly or indirectly) and what of the humans that get in contact with the poisons or even those that might eat the birds killed in the exercise?

The poisoning saga in Kenya is terrifying, especially when the true knowledge of the deadly implications of the pesticides being used to kill vermin and wild animals is sat on squarely by the relevant authorities so that for some reason the public does not get to it. This looks bad especially when the whole situation appears to take full advantage of public ignorance and employing otherwise deadly chemicals that qualify to be termed poisonous. Sometimes, sharing the knowledge on these chemicals is worthwhile and may enhance livelihood security.

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