Stop Wildlife Poisoning

Category: carbofuran | Date: Jul 30 2009 | By: Martin Odino

Dear readers, the summary on the post 4 points on carbofuran poisoning in humans revealed intentional poisoning in humans as the mode by which human mortalities occured. So is the case for lion poisoning and likewise bird poisoning. But EPA spells that even proper use of the pesticide is harmful especially to children, hence the ongoing process to ban it from the USA “in a move to protect people, especially children, from dietary risk.”

The lab analysts that gave me the facts in the post, 4 points on carbofuran poisoning in humans did not understand why the people feeding on Furadan-poisoned birds in Bunyala (and Mwea) region were not dying. First and foremost, there has been no medical research on the effects of the poison on the people in the areas. Data on human poisoning let alone precision of actual responsible poison is retreavable from the poorly staffed and equiped health centres in the area. We therefore concluded that most likely the deaths occur but are never reported.

But locals and the 2 doctors who also own pharmacies and whom I got to talk to in Bunyala agree there are a couple of mortality cases that occur in the area are certainly poisoning cases. When asked which poison they thought was responsible, they gave a number but named Furadan first; asked what they thought the most common and probable poison used to kill the humans was, they picked on Furadan. Both doctors admit they have no proper records where they have recorded the poisoning cases. For another reason, locals are lacking in liquid cash, though their livestock assets are modest and apparently they save these for last though this is not well illustrated given the high rate of illiteracy and long-illness deaths. The locals therefore will tend to self-treat or strain their immunities to fight without medicinal back up. In such cases, people will try giving the poisoned fellow milk at best otherwise they pass on without medical intervention. Even in death, post-mortems are never carried out let alone being known. The berieved then cling onto the corpse of their beloved one for fear of superstitious practices being performed on their own in case the body has to be taken to hospital for medical examination in which case the dead would remain to haunt them. And so, if Furadan was the cause of death the evidence goes with the dead to the grave.

At the lab, one analyst tried explaining the phenomenon of ‘no observable’ intoxication in humans in Bunyala. He explained that the poison once ingested by a bird goes to the brain before going elsewhere in the body. I wish I understood that but I had to be contented with the path of poison travel as first to the brain. Of course the alimentary canal is tainted with the poison as well. Consumers of the poisoned birds normally get rid of the head, legs and entrails therefore lower the quantity that will be consumed. Nonetheless, the expert said that in this poison’s killing path, the poison does spread. Further, during cooking, the poison is concentrated citing that they use heat to concentrate Furadan in samples to be analysed in the lab for carbofuran. The lttle Furadan in the bird’s meat is therfore consumed after being concentrated even though it may not attain the lethal amount!

Poisoned pigeon in Bunyala: the bird had been picked by 3 boys having died out of the poacher’s poisoning range. Poison is concentrated in the brain and gut.

In the post, Furadan Everywhere, I tried to explain how the Furadan used in Bunyala gets ‘everywhere’ inclusive of domestic water sources. In EPA’s Tolerance Revocation, “EPA completed action to revoke existing carbofuran tolerances (residue limits in food) due to unacceptable dietary risks, especially to children, from consuming a combination of food and water with carbofuran residues.” I think people in Bunyala are consuming this combination. More is that a majority are youngsters (children) with rice being one of the immediate post-weaning foodstuffs that they have to get used to eating.

Kids giving mummy a helping hand with the rice; will they grow to be strong?will they survive? There might just be Furadan residues in the rice that cou;d cause intoxication.

Rice cultivation is inseparable from Furadan use; it is the media by which Furadan found its way to Bunyala. The supply was so gross that the Furadan supplied at planting time from the time the seedlings are developing in the seedbeds remains to be used in vegetable shambas (gardens), poisoning birds and for other crude uses suh as being put on wounds to keep off flies. Those who have tried the latter say the flies drop dead.And it is also the efficient way to commit suicide or kill another person(s) who you are not in very good terms with! As far as rice production is concerned, the rice grown in the fields is as good as a cash crop only not sold by the farmers: the irrigation board claims its lion’s share for the planting inputs that were provided to the farmers once the rice matures and is harvested. Some however remains with the farmers who make it almost an all-meals constituent. In the morning it is taken with tea, at lunch time it may be taken with legumes, mostly beans and in the evening with poisoned bird stew or skipped to be eaten the next day at the frequency of at least 2 meals a day. But EPA says that Furadan even when used as a nematicide correctly in such a manner as rice production, still its residues will remain in the food. So the Banyalas and Luos in satiating their hunger are lodging an intoxicant in their bodies with those unable to survive passing on quietly.

It is a long string of deaths due to Furadan: animal bait(snails, small fish, grasshoppers, locusts,), birds and humans. Elsewhere our majestic big cats are at its mercy.

Please keep reading.

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